25. January 2016

A Genome-wide CRISPR Screen Identifies NEK7 as an Essential Component of NLRP3 Inflammasome Activation Jonathan Schmid-Burgk and colleagues present new data on the identification of NEK7

The mechanisms of NLRP3 activation are still poorly understood. Jonathan Schmid-Burgk and colleagues present new data on the identification of NEK7, which specifically functions upstream of NLRP3 activation. NEK7 was identified in an unbiased genetic screening approach, which employed the CRISPR technology to identify macrophages that were rendered defective in NLRP3 signal transduction.

Institute for Clinical Chemistry and Clinical Pharmacology University and University Hospital Bonn
Institute for Clinical Chemistry and Clinical Pharmacology University and University Hospital Bonn © Elmar Endl, Uni Bonn
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Description of a genome-wide CRISPR/Cas9 screen in immortalized mouse macrophages aiming at the unbiased identification of gene products involved in Nlrp3 inflammasome activation. We employed a FACS-based screen for Nlrp3-dependent cell death, using the ionophoric compound nigericin as a potassium efflux-inducing stimulus.

FACS-based screening strategy to identify components upstream of Nlrp3 inflammasome activation. A, under steady state conditions Nlrp3 inflammasome activation requires a priming signal (signal 1) that upregulates Nlrp3 expression (left panel). To overcome this requirement, cells stably expressing Nlrp3 were used (right panel). These cells directly respond to nigericin (signal 2) without a previous priming signal. B, Following transduction with a gRNA (additionally encoding for GFP), cells are stimulated with nigericin or left untreated. Cells are then stained with PI and subjected to FACS analysis. Transduced cells that are alive are GFP positive and PI negative, whereas dead cells lose their GFP positivity, while acquiring a PI signal.

Flow Cytometry Core Facility at the Medical Faculty of the University Bonn

Schmid-Burgk, J. L. et al. A Genome-wide CRISPR (Clustered Regularly Interspaced Short Palindromic Repeats) Screen Identifies NEK7 as an Essential Component of NLRP3 Inflammasome Activation. J Biol Chem 291, 103–109 (2016).

Prof. Jonathan Schmid-Burgk
Institute for Clinical Chemistry and Clinical Pharmacology
University and University Hospital Bonn
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